Xiao Yiyi,Zhao Ziyang,Tong Yanlu,et al.Experimental study of bile duct injury caused by cholesterol-activated CD177+ neutrophils in biliary atresia[J].Journal of Clinical Pediatric Surgery,,22():782-789.[doi:10.3760/cma.j.cn101785-202303064-016]
Click Copy

Experimental study of bile duct injury caused by cholesterol-activated CD177+ neutrophils in biliary atresia

References:

[1] Lakshminarayanan B, Davenport M.Biliary atresia:a comprehensive review[J].J Autoimmun, 2016, 73:1-9.DOI:10.1016/j.jaut.2016.06.005.
[2] Schreiber RA.Newborn screening for biliary atresia[J].JAMA, 2020, 323(12):1137-1138.DOI:10.1001/jama.2020.2727.
[3] 詹江华, 孙溶涓, 王智茹, 等.日本《胆道闭锁临床实践指南》解读[J].临床小儿外科杂志, 2023, 22(3):205-210.DOI:10.3760/cma.j.cn101785-202212025-002. Zhan JH, Sun RJ, Wang ZR, et al.Interpretations of Clinical Practice Guidelines for Biliary Atresia in Japan[J].J Clin Ped Sur, 2023, 22(3):205-210.DOI:10.3760/cma.j.cn101785-202212025-002.
[4] Mack CL, Falta MT, Sullivan AK, et al.Oligoclonal expansions of CD4+ and CD8+ T-cells in the target organ of patients with biliary atresia[J].Gastroenterology, 2007, 133(1):278-287.DOI:10.1053/j.gastro.2007.04.032.
[5] Zhang Y, Zhou L, Gu GX, et al.CXCL8highinflammatory B cells in the peripheral blood of patients with biliary atresia are involved in disease progression[J].Immunol Cell Biol, 2020, 98(8):682-692.DOI:10.1111/imcb.12366.
[6] Miethke AG, Saxena V, Shivakumar P, et al.Post-natal paucity of regulatory T cells and control of NK cell activation in experimental biliary atresia[J].J Hepatol, 2010, 52(5):718-726.DOI:10.1016/j.jhep.2009.12.027.
[7] 阿里木江·阿不都热依木, 林峰, 王皓洁, 等.Notch信号通路活化协同巨噬细胞对胆道闭锁肝纤维化的作用研究[J].临床小儿外科杂志, 2021, 20(4):376-381.DOI:10.12260/lcxewkzz.2021.04.014. Abudureyimu ALMJ, Lin F, Wang HJ, et al.Activation of notch signaling pathway collaborated with macrophages for promoting liver fibrosis in biliary atresia[J].J Clin Ped Sur, 2021, 20(4):376-381.DOI:10.12260/lcxewkzz.2021.04.014.
[8] Zhang RZ, Su L, Fu M, et al.CD177+ cells produce neutrophil extracellular traps that promote biliary atresia[J].J Hepatol, 2022, 77(5):1299-1310.DOI:10.1016/j.jhep.2022.06.015.
[9] Manzel A, Muller DN, Hafler DA, et al.Role of "western diet" in inflammatory autoimmune diseases[J].Curr Allergy Asthma Rep, 2014, 14(1):404.DOI:10.1007/s11882-013-0404-6.
[10] Chen YC, Tung KY, Tsai CH, et al.Lipid profiles in children with and without asthma:interaction of asthma and obesity on hyperlipidemia[J].Diabetes Metab Syndr, 2013, 7(1):20-25.DOI:10.1016/j.dsx.2013.02.026.
[11] Chen XY, Yin QH, Ma L, et al.The role of cholesterol homeostasis in diabetic kidney disease[J].Curr Med Chem, 2021, 28(36):7413-7426.DOI:10.2174/0929867328666210419132807.
[12] Janoudi A, Shamoun FE, Kalavakunta JK, et al.Cholesterol crystal induced arterial inflammation and destabilization of atherosclerotic plaque[J].Eur Heart J, 2016, 37(25):1959-1967.DOI:10.1093/eurheartj/ehv653.
[13] Kim MC, Borcherding N, Ahmed KK, et al.CD177 modulates the function and homeostasis of tumor-infiltrating regulatory T cells[J].Nat Commun, 2021, 12(1):5764.DOI:10.1038/s41467-021-26091-4.
[14] Hu N, Mora-Jensen H, Theilgaard-M?nch K, et al.Differential expression of granulopoiesis related genes in neutrophil subsets distinguished by membrane expression of CD177[J].PLoS One, 2014, 9(6):e99671.DOI:10.1371/journal.pone.0099671.
[15] Warnatsch A, Ioannou M, Wang Q, et al.Inflammation.neutrophil extracellular traps license macrophages for cytokine production in atherosclerosis[J].Science, 2015, 349(6245):316-320.DOI:10.1126/science.aaa8064.
[16] Drechsler M, Megens RTA, van Zandvoort M, et al.Hyperlipidemia-triggered neutrophilia promotes early atherosclerosis[J].Circulation, 2010, 122(18):1837-1845.DOI:10.1161/CIRCULATIONAHA.110.961714.
[17] Lood C, Blanco LP, Purmalek MM, et al.Neutrophil extracellular traps enriched in oxidized mitochondrial DNA are interferogenic and contribute to lupus-like disease[J].Nat Med, 2016, 22(2):146-153.DOI:10.1038/nm.4027.
[18] Dan Dunn J, Alvarez LA, Zhang XZ, et al.Reactive oxygen species and mitochondria:a nexus of cellular homeostasis[J].Redox Biol, 2015, 6:472-485.DOI:10.1016/j.redox.2015.09.005.

Memo

收稿日期:2023-03-29。
基金项目:国家自然科学基金面上项目(81974056,82271750);广州市科技计划项目(202206080002)
通讯作者:张锐忠,Email:cowboy2006@163.com

Last Update: 1900-01-01