[1]刘红综述,马洪审校.先天性肾积水肾脏纤维化机制的研究进展[J].临床小儿外科杂志,2013,12(03):240-242.[doi:10.3969/j.issn.1671—6353.2013.03.025]
点击复制

先天性肾积水肾脏纤维化机制的研究进展

参考文献/References:

1张国贤,文建国,王庆伟,等.尿液AQP2在先天性肾积水患儿中的检测和意义[J].临床小儿外科杂志,2008,7(5):3—6.
2Gaedeke J,Peters H,Noble NA,et al.Angiotensin II,TGFβ and renal fibrosis[J].Contrib Nephrol,2001,(135):153—160.
3毛永忠,汤绍涛,阮庆兰,等.胆道闭锁肝脏组织MMP—2、TGF—β1的表达及意义[J].临床小儿外科杂志,2007,6(3):6—9.
4Liu Y.New insights into epithelial-mesenchymal transition in kidney fibrosis[J]. JAM Soc Nephrol,2010,21(2):212—222.
5ChiaoYin Sun,ShihChung Chang,MaiSzu Wu.Uremic Toxins Induce Kidney Fibrosis by Activating Intrarenal ReninAngiotensinAldosterone Sstem Associated EpithelialtoMesenchymal Transition[J].PLoS One,2012,7(3): 34026.
6Ye S, ZhongH, Yanamadala S,et al. Oxidative stressmediates the stimulation of sympathetic nerve activity in the phenol renal Injury model ofhypertension[J].Hypertension, 2006,48(2):309—315.
7Kajbafzadeh AM, Payabvash S, Salmasi AH,et al. Smooth muscle cell apoptosis and defective neural development in congenital ureteropelvic junction obstruction[J].J Urol, 2006, 176(2): 718—723.
8Trachtman H, Futterweit S,Garg P, et al. Nitric oxide stimulates the activity of a 72—kDa neutral matrix metalloproteinase in cultured rat mesangial cells[J].Biochem Biophys Res Commun, 1996, 218(3): 704—708.
9Gupta A,Sharma S,Chopra K.Reversal of ironinduced nephrotoxicity in rats bymolsidomine, a nitric oxide donor[J]. Food Chem Toxico,2008,46(2): 537—543.
10陈腾锋,谢庆祥,张跃红.肾脏纤维化机制的研究进展[J].国际泌尿系统杂志,2011,31(3):404—408.
11Li SD, Yuan BL. Effect of NO on acute severe hepatic injury induced by integrative LPS and GaIN [J].China Journal of Modern Medicine,2005, 15(11): 1609-1612. Chinese.
12Yoo KH, Thornhill BA, Forbes MS, et al. Inducible nitric oxide synthase modulates hydronephrosis following partial or complete unilateral ureteral obstruction in the neonatal mouse [J]. Am J Physiol Renal Physiol, 2010, 298(1): F62—71.
13辛冰牧,杨红振,胡卓伟. 肾纤维化发病机制及治疗学研究进展[J].国际药学研究杂志, 2008, 35(5): 349—353.
14Hatsukari I, Hitosugi N, Dinda A, et al. Morphin modulates monocytemacrophage conversion phase [J]. Cell Immunol,2006, 239(1): 41—48.
15Metcalfe PD,Leslie JA,Campbell MT,et al.Testosterone exacerbates obstructive renal injury by stimulating TNFalpha production and increasing proapoptotic and profibrotic signaling〔J〕. Am J Physiol Endocrinol Metab,2008; 294( 2) : E435—43.
16高绪霞,黄海长,李晓玫.成肌纤维细胞在纤维化过程中的作用及其调控.生理科学进展[J].2007;38(1):78—80.
17Bohle A,MullerGA,WehrmannM,et al. Pathogenesis of chronicrenal failure in the primary glomerulopathies, renal vasclopathies,and chronic interstitial nephritides[J].Kidney Int, 1996, 49(1):S2—S9.
18周鸿敏,袁继炎,王涛.先天性肾发育不良TGF—β1和α—SMA表达的研究[J].临床小儿外科杂志,2002,1(3):178—180.
19Zeisberg EM,Potenta SE,Sugimoto H,et al.Fibroblastd inkifney fibrosis emerge via endothelialto-mesenchymal tran-sitio[J].Journal of the American Society of Nephrology, 2008,19(12):2282—2287.
20Johnson TS,Haylor JL,Thomas GL,et al.Matrix metalloprotein ases and their inhibitions in experimental renals carring[J].Exp Nephrol,2002,10(3):182—195.
21Wharram BL,Gora LM,Wiggins JE,et al.Podocyte depletion causes glomerulosclerosis :diphtheria toxininduced podocyte depletion in rats expressing human diphtheria toxin receptor transgene[J].J AM Soc Nephrel,2005,16(10):2941—2952.
22Woo M, Hakem R, Soengas MS,et al. Essential contribution of caspase3/CPP32 to apoptosis and its associated nuclear changes[J]. Genes Dev,1998,12(6):806—819.
23Yang B, Nahas ME, Thomas GL,et al. Caspase-3 and apoptosisin experimental chronic renal scarring [J].Kidney Int,2001,60(6):1765—1776.
24Habek D, Kulas T, Selthofer R, et al. Successful of the BLynch compression suture in the management of massive opstpartum hemorrhage: case reports and review[J].ArchGynecol Obstet, 2006, 273(5): 307—309.

更新日期/Last Update: 2013-06-30